whole mouse genome microarray comparisons (Agilent technologies)
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whole mouse genome microarray comparisons
Whole Mouse Genome Microarray Comparisons, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray+comparisons/pm21320871-199-1-0
Average 86 stars, based on 1 article reviews
Whole Mouse Genome Microarray Comparisons, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray+comparisons/pm21320871-199-1-0
Average 86 stars, based on 1 article reviews
whole mouse genome microarray comparisons - by Bioz Stars,
2026-11
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Hybridization:Article Title: Genetic alterations determine chemotherapy resistance in childhood T-ALL: modelling in stage-specific cell lines and correlation with diagnostic patient samples. Article Snippet: Treatment failure in T-cell acute lymphoblastic leukaemia (T-ALL) occurs when leukaemic blasts acquire resistance to chemotherapeutic agents (Pui et al, 2002).. T-ALL is a biologically and clinically distinct disease entity that is associated with clinically aggressive features and poor treatment outcomes (Uckun et al, 1998; Pui & Evans, 2006).. Although 95% of children and young adults currently treated for T-ALL enter into a first remission, approximately 25% of patients relapse for reasons that remain unclear (Kanerva et al, 2001; Tafuri et al, 2002). Microarray:Article Title: Genetic alterations determine chemotherapy resistance in childhood T-ALL: modelling in stage-specific cell lines and correlation with diagnostic patient samples. Article Snippet: Treatment failure in T-cell acute lymphoblastic leukaemia (T-ALL) occurs when leukaemic blasts acquire resistance to chemotherapeutic agents (Pui et al, 2002).. T-ALL is a biologically and clinically distinct disease entity that is associated with clinically aggressive features and poor treatment outcomes (Uckun et al, 1998; Pui & Evans, 2006).. Although 95% of children and young adults currently treated for T-ALL enter into a first remission, approximately 25% of patients relapse for reasons that remain unclear (Kanerva et al, 2001; Tafuri et al, 2002). Article Title: Puromycin-sensitive aminopeptidase (PSA/NPEPPS) impedes development of neuropathology in hPSA/TAU(P301L) double-transgenic mice. Article Snippet: The concentration and quality of RNA were determined using the Nanodrop spectrophotometer and confirmed on the Agilent Bioanalyzer. .. 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Supplementary Table 1 presents such comparisons with data obtained through Article Title: Deciphering potential vascularization factors of on-chip co-cultured hiPSC-derived cerebral organoids. Article Snippet: The dye incorporation efficiency was detected using a NanoDrop One/One spectrophotometer (Thermo Fisher Scientific). .. Agilent Transgenic Assay:Article Title: Puromycin-sensitive aminopeptidase (PSA/NPEPPS) impedes development of neuropathology in hPSA/TAU(P301L) double-transgenic mice. Article Snippet: The concentration and quality of RNA were determined using the Nanodrop spectrophotometer and confirmed on the Agilent Bioanalyzer. .. Agilent Control:Article Title: Puromycin-sensitive aminopeptidase (PSA/NPEPPS) impedes development of neuropathology in hPSA/TAU(P301L) double-transgenic mice. Article Snippet: The concentration and quality of RNA were determined using the Nanodrop spectrophotometer and confirmed on the Agilent Bioanalyzer. .. Agilent Article Title: Deciphering potential vascularization factors of on-chip co-cultured hiPSC-derived cerebral organoids. Article Snippet: The dye incorporation efficiency was detected using a NanoDrop One/One spectrophotometer (Thermo Fisher Scientific). .. Agilent Comparison:Article Title: Puromycin-sensitive aminopeptidase (PSA/NPEPPS) impedes development of neuropathology in hPSA/TAU(P301L) double-transgenic mice. Article Snippet: The concentration and quality of RNA were determined using the Nanodrop spectrophotometer and confirmed on the Agilent Bioanalyzer. .. Agilent Article Title: Deciphering potential vascularization factors of on-chip co-cultured hiPSC-derived cerebral organoids. Article Snippet: The dye incorporation efficiency was detected using a NanoDrop One/One spectrophotometer (Thermo Fisher Scientific). .. Agilent Extraction:Article Title: High-resolution analysis of the m-xylene/toluene biodegradation subtranscriptome of Pseudomonas putida mt-2. 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![Figure 4. PCSK6 is the most significantly upregulated proprotein convertase (PC) in symptomatic carotid plaques. Analy- ses of n=127 microarray profiles between carotid plaques (CP) and normal controls (iliac arteries [IA]) show significant down- regulation of Furin and PCSK5 in plaque tissue, upregulation of PCSK6 and PCSK7, whereas PCSK9 and PCSK2 did not exhibit significant variation in expression (A). Posi- tive correlation was found between expres- sion of PCSK6 and PCSK9, PCSK2 and PCSK7, whereas negative was observed with PCSK5 (B). Several PCSK6 isoforms recognized by different Affymetrix probes were significantly upregulated in plaques compared with controls, but most highly the isoform coding for secreted protein (C). This isoform was the only one with signifi- cantly higher expression in comparison between symptomatic (s) and asymptom- atic (as) plaques (D). Results were con- firmed by quantitative real-time polymerase chain reaction (qRT-PCR) analyses for expression of secreted PCSK6 isoform in a separate cohort of n=233 carotid plaques (E and F). Micorarray data expressed as log2 value and qRT-PCR data as fold change compared with control. Data in A, C, and D show mean with SD, whereas median with interquartile range is depicted in E and F. ER indicates endoplasmatic reticulum.](https://doi-unpaywalled-images-cdn.bioz.com/3104/10__1161_slash_atvbaha__113__301743/10__1161_slash_atvbaha__113__301743____page7_image1.jpg)